By G. Vidya Sagar
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Additional resources for MCQs in Pharmacology
D 200. c 201. b 202. a 203. d 204. a 205. d 206. d 207. b 208. c 209. b 210. b 211. b 212. c 213. a 214. b 215. b 216. c 217. d 218. c 219. a 220. a 221. c 222. c 223. d 224. d 225. d 226. a 227. b 228. b 229. c 230. b 231. a 232. b 233. e 234. d 235. a 236. e 237. a 238. d 239. e 240. a 241. b 242. d 243. a 244. d 245. d 246. 5 247. c 248. e 249. c 250. c 251. c 252. a 253. d 254. b 255. d 256. b 257. d 258. f 259. c 260. b 261. a 262. d 263. c 264. b 265. c 266. d 267. c 268. d 269. d 270. e 271.
A 65. c 78. c 110. b 125. c 145. d 161. d because of enhanced disassociation weak acids are excreted faster in basic (not acidic) urine. Chemical antagonism does not involve any receptors. Protamine is a positively charged protein at physiological pH and thus antagonizes the effects of Heparin which is negatively charged at physiological pH. Facilitated diffusion differs from active transport in that it does not require energy source and it carries the transport in the direction of electrochemical gradient.
Two drugs binding to the same receptor is an example of competitive antagonism and effect of one drug can be decreased by increasing the concentration of other drug. Receptors for steroid hormones are intracellular DNA – binding proteins, which regulate gene transcription. g. nicotinic acetylcholine receptor. e. it is made up of five poly-peptide subunits. In G-protein coupled receptors, agonist-receptor complex enhances GTP binding to the α subunit, mainly by dissociating already bound GDP. The termination of agonist-receptor coupling in a G-protein-coupled receptor is because of conversion of GTP to GDP by a GTPase that is intrinsic to α subunit.