By Christopher P. Cannon
Brigham and Women's medical institution, Boston, MA. offers clinicians and citizens with a present evaluate of some of the advances within the pathophysiology, analysis, and administration of sufferers with acute coronary syndromes. positive factors new assessments, brokers, ways, and regimens. earlier version: c1999. DNLM: Coronary Disease--therapy.
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Extra resources for Malformations of the Nervous System Handbook of Clinical Neurology Series
Moderate defects, including unilateral or bilateral clefts, midface hypoplasia and moderate hypotelorism occurred in 15%. Mild malformations, including mild hypotelorism (Fig. 10E) and single maxillary central incisor (Fig. 10F), were seen in 41%. A total of 27% of our patient cohort had no facial dysmorphisms. , 2002). Thus the more severe the facial malformation the more likely it was that the child would have a severe type of HPE. , 2002). For example, three of 21 alobar patients had no discernible facial dysmorphism and two of 17 lobar HPE patients had premaxillary agenesis with median cleft.
Even though in some of these cases, fusion of the thalamic nuclei may be identified, in absence of some degree of neocortical nonseparation, these cases should not be classified as HPE. Nevertheless, cases with thalamic fusion may share similar pathogenetic mechanisms involved in HPE and further studies are needed to understand the boundaries and continuities of midline malformations. Using three-dimensional imaging with reconstruction, detailed topological studies have been performed 26 J. S. , 2003, 2004).
Hum Mutat 16: 99–108. Wassif CA, Maslen C, Kachilele-Linjewile S, et al. (1998). Mutations in the human sterol d7-reductase gene at 11q12–13 cause Smith–Lemli–Opitz syndrome. Am J Hum Genet 63: 55–62. Watanabe K, Hara K, Iwase K (1976). The evolution of neurophysiological features in holoprosencephaly. Neuropa¨diatrie 7: 19–41. 37 Wong AM, Bilaniuk LT, Ng KK, et al. (2005). Lobar holoprosencephaly: prenatal MR diagnosis with postnatal MR correlation. Prenat Diagn 25: 296–299. Yakovlev PI (1959).