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By N. G. Conradi (auth.), Prof. Dr. Kurt Jellinger, Prof. Dr. Filippo Gullotta, Prof. Dr. Miroslav Mossakowski (eds.)

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Read or Download Experimental and Clinical Neuropathology: Proceedings of the First European Neuropathology Meeting, Vienna, May 6–8, 1980 PDF

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Additional resources for Experimental and Clinical Neuropathology: Proceedings of the First European Neuropathology Meeting, Vienna, May 6–8, 1980

Example text

Rehncrona S, Rosen I, SOderfeldt B, Siesjo BK (in press) The influence of lactate production in the ischemic brain on the capacity for postischemic recovery. D. Department of Pathology University of Turku SF-20520 Turku 52 Finland Acta N europathol (Berl) Supp!. VII, 23-25 © Springer-Verlag 1981 Acceleration Induced Head Injury in the Monkey. I. A. Gennarellil, IH. I. Graham2 1 Department of Neurosurgery, University of Pennsylvania, Philadelphia, Pa, USA, 2 Department of Neuropathology, University of Glasgow, Glasgow, Great Britain acceleration could be studied in isolation to allow testing of the hypothesis that angular acceleration is an important mechanism for producing the physiological and pathological consequences seen in closed head injury.

Thus the effect of Materials and Methods By varying the magnitude of angular acceleration, this system was capable of producing graded injuries from subconcussive to instantaneously lethal. In order to categorize the clinical responses of the animals to the injury, an experimental trauma severity (ETS) scale was devised (Fig. 1). This has proved to be a useful shorthand means of scaling the injury severity and is based on observed changes in blood pressure, heart rate and respiration; the level of conscious- 23 Experimental Trauma Severity (ETS) o 2 3 4 5 6 Bradycardia Apnea Lass of cameal reflex Loss of corneal reflex > 30 sec Cerebral death <6hrs Immediate death Fig.

In ETS grade 0 there was no evidence that the brain had been disturbed by the acceleration. The animal was completely normal behaviorally and neurologically immediately after the acceleration. In ETS grade 1 there were changes in the SAP and heart rate, but the animals were behaviorally and neurologically normal. In ETS grade 2 a brief period of apnea accompanied the alterations in SAP and heart rate. The period of apnea was generally followed by respiratory irregularities. The animals were not behaviorally unconscious.

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